<<
>>

1.3.5 Treponema denticola

Treponema denticola it is a Gram-negative bacterium, obligate anaerob. Treponema denticola promotes production metalloproteinaz from PMJAL,

Causing a destruction of intercellular substance of a connecting tissue.

T. denticola can aggljutinirovat and lyse erythrocytes. The spirochete of oral cavity Treponema denticola does not get into live epitheliocytes, and induces a depolymerization and rearanzhirovku aktinovyh microfilaments, along with weakening of fastening of epitheliocytes. Superficial fiber T. denticola can move to a membrane of an epitheliocyte with its the subsequent depoljarizatsiej and formation of ionic channels. As a result of such attack of function of epitheliocytes are broken, and at HGP in a material it is found out much treponem [78]. Ability of it parodontopatogena is proved to activate macrophages which in turn allocate the substances promoting rezorbtsii of collagen (nitrous oxide and cytokines). T. denticola against the broken and normal function of neutrophils causes the deep centres of a lesion [206]. Тreponema denticola it can be attached to an endothelium, contact cells on all their extent. It forms units with P. gingivalis and Fusobacterium spp., that can matter for formation of a tooth plaque, and also for a food

Bacteria.

<< | >>
Scientific source YELISEYEVA Anna Fyodorovna. SOCHETANNOE LESION PARODONTA And CARDIOVASCULAR SYSTEM, CLINICO-MORPHOLOGICAL And MICROBIOLOGICAL RESEARCH. The dissertation on competition of a scientific degree of the candidate of medical sciences. St.-Petersburg – 2014. 2014

Other medical related information 1.3.5 Treponema denticola:

  1. the Table of contents
  2. INTRODUCTION
  3. 1.3 Role of the microbic factor in development chronic generalised parodontita
  4. 1.3.5 Treponema denticola
  5. 2.2 Methods of research
  6. 2.3.1 Molekuljarno-microbiological methods
  7. the Diagnosis
  8. Clinical observation 2
  9. Clinical observation 3
  10. Clinical observation 4
  11. Clinical observation 5
  12. THE CONCLUSION
  13. CONCLUSIONS